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Mechanism of action: naloxone hydrochloride is an opioid antagonist with greatest affinity for the mu receptor. It acts by competing for the mu, kappa, and sigma opiate receptor sites in the CNS.
Crash Cart, Emergency Drug
Mechanism of action: Dextrose solution provides a source of water and carbohydrate (340 kcal/L). The simple carbohydrate may minimize liver glycogen depletion and provide a protein-sparing action.
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Mechanism of action: An organic nitrate, is a vasodilating agent that relieves tension on vascular smooth muscle and dilates peripheral veins and arteries.
Crash Cart, Emergency Drug
Mechanism of action: Aspirin is a more potent inhibitor of both prostaglandin synthesis and platelet aggregation than its other salicylic derivatives due to the acetyl group on the aspirin molecule, which irreversibly inactivates cyclooxygenase via acetylation.
Crash Cart, Emergency Drug
Mechanism of Action: Strong beta1- and alpha-adrenergic effects and moderate beta2 effects, which increase cardiac output and heart rate, decrease renal perfusion and PVR, and cause variable BP effects
Crash Cart, Emergency Drug, High Alert
Mechanism of Action: Depresses CNS, blocks peripheral neuromuscular transmission, produces anticonvulsant effects; decreases amount of acetylcholine released at end-plate by motor nerve impulse.
Crash Cart, Emergency Drug, High Alert
Mechanism of Action: Strong alpha-adrenergic effects, which cause an increase in cardio output and HR, a decrease in renal perfusion and PVR, and a variable effect on BP, resulting in systemic vasoconstriction and increased vascular permeability
Crash Cart, Emergency Drug, High Alert
Mechanism of Action: Strong beta1 and weak beta2/alpha effects, resulting in increased cardiac output, blood pressure, and heart rate, as well as decreased peripheral vascular resistance
Crash Cart, Emergency Drug, High Alert
Mechanism of Action: Parenteral dextrose is oxidized to carbon dioxide and water, and provides 3.4 cal/g of d-glucose
Crash Cart, Emergency Drug, High Alert
Drug
Naloxone
Mechanism of action: naloxone hydrochloride is an opioid antagonist with greatest affinity for the mu receptor. It acts by competing for the mu, kappa, and sigma opiate receptor sites in the CNS.
Drug
Dextrose
Mechanism of action: Dextrose solution provides a source of water and carbohydrate (340 kcal/L). The simple carbohydrate may minimize liver glycogen depletion and provide a protein-sparing action.
Drug
Nitroglycerin
Mechanism of action: An organic nitrate, is a vasodilating agent that relieves tension on vascular smooth muscle and dilates peripheral veins and arteries.
Drug
Aspirin
Mechanism of action: Aspirin is a more potent inhibitor of both prostaglandin synthesis and platelet aggregation than its other salicylic derivatives due to the acetyl group on the aspirin molecule, which irreversibly inactivates cyclooxygenase via acetylation.
Drug
Norepinephrine
Mechanism of Action: Strong beta1- and alpha-adrenergic effects and moderate beta2 effects, which increase cardiac output and heart rate, decrease renal perfusion and PVR, and cause variable BP effects
Drug
Magnesium sulfate
Mechanism of Action: Depresses CNS, blocks peripheral neuromuscular transmission, produces anticonvulsant effects; decreases amount of acetylcholine released at end-plate by motor nerve impulse.
Drug
Epinephrine
Mechanism of Action: Strong alpha-adrenergic effects, which cause an increase in cardio output and HR, a decrease in renal perfusion and PVR, and a variable effect on BP, resulting in systemic vasoconstriction and increased vascular permeability
Drug
Dobutamine
Mechanism of Action: Strong beta1 and weak beta2/alpha effects, resulting in increased cardiac output, blood pressure, and heart rate, as well as decreased peripheral vascular resistance
Drug
Dextrose 50%
Mechanism of Action: Parenteral dextrose is oxidized to carbon dioxide and water, and provides 3.4 cal/g of d-glucose
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